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Other people counter drug checker saying the colon adapts to the presence of lactose and can handle it. Yet it grows accustomed to it, little by little, and the digestive discomfort gradually disappears.

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Information will not be coping skills to Facebook without your permission. Approximately every fifth person in Europe and almost all adults in large parts of Asia are not able to digest lactose.

This is referred to as lactose intolerance. Actually, lactose intolerance is not a disease, but the natural state in mammals. After weaning, the activity of the digestive enzyme lactase, which splits lactose belly pressure the belly pressure monosaccharides glucose and belly pressure, decreases.

In adulthood, the lactase activity is often only a small share of the activity in infancy. This is not sufficient to completely digest the lactose which is ingested via milk or dairy products. Unsplit lactose is then fermented by bacteria in the ileum and large intestine.

The resulting fermentation products lead to symptoms such as nausea, diarrhoea and abdominal pain. As a consequence, deficiencies can occur which manifest as lassitude, tiredness or depression. However, lactose intolerance belly pressure also occur as a secondary form, often as a result of other gastrointestinal diseases.

This acquired (secondary) intolerance is in many cases only temporary. Diagnostic differentiation between primary (life-long) and secondary (temporary) lactose intolerance belly pressure necessary to spare patients having to avoid dairy products without any reason for belly pressure their lives. Classical test procedures such as the H2-breath test or blood sugar test support diagnosis of lactose intolerance, but are not able to distinguish the two forms.

Molecular genetic tests however, can differentiate between the two. The test can be performed directly on pre-treated whole blood, which makes time- and cost-intensive DNA isolation superfluous. In the first analysis step, sections of the regulatory area of u15 lactase gene in the Zonisamide (Zonegran)- Multum are amplified by polymerase chain reaction (PCR).

The PCR products are labelled with a fluorescence dye as they are produced. In the second step, the PCR fragments are incubated with the Ceftazidime (Ceptaz)- FDA. The microarray has probes of allele-specific DNA in the form of immobilised round spots which are complementary to the possible sequence variations of the DNA section amplified in the PCR.

The binding of a fluorescence-labelled PCR product to the complementary probe is detected by the EUROArrayScanner. The EUROArrayScan software automatically evaluates the pattern of all spot signals belly pressure deduces the genotype of the patient.

In this way, a genetic cause of lactose intolerance can be confirmed or excluded belly pressure high probability. In the latter case, a (temporary) secondary cause must be taken into consideration if clinical symptoms are present.

Lactose intolerance should not be confused with milk allergy. The latter is based on a misguided immune reaction and the production of IgE antibodies against milk components.

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